Cookies on this website

We use cookies to ensure that we give you the best experience on our website. If you click 'Accept all cookies' we'll assume that you are happy to receive all cookies and you won't see this message again. If you click 'Reject all non-essential cookies' only necessary cookies providing core functionality such as security, network management, and accessibility will be enabled. Click 'Find out more' for information on how to change your cookie settings.

The skin is the most common target organ affected by graft-versus-host disease (GVHD), with severity and response to therapy representing important predictors of patient survival. Although many of the initiating events in GVHD pathogenesis have been defined, less is known about why treatment resistance occurs or why there is often a permanent failure to restore tissue homeostasis. Emerging data suggest that the unique immune microenvironment in the skin is responsible for defining location- and context-specific mechanisms of injury that are distinct from those involved in other target organs. In this review, we address recent advances in our understanding of GVHD biology in the skin and outline the new research themes that will ultimately enable design of precision therapies.

Original publication

DOI

10.3389/fimmu.2018.00963

Type

Journal article

Journal

Front Immunol

Publication Date

2018

Volume

9

Keywords

B cells, T cells, antigen-presenting cells, cutaneous graft-versus-host disease, microbiome, pathophysiology, repair mechanisms, Animals, B-Lymphocytes, Cellular Microenvironment, Cytokines, Graft vs Host Disease, Hematopoietic Stem Cell Transplantation, Humans, Mice, Microbiota, Skin Diseases, T-Lymphocytes, Transplantation, Homologous