Cookies on this website

We use cookies to ensure that we give you the best experience on our website. If you click 'Accept all cookies' we'll assume that you are happy to receive all cookies and you won't see this message again. If you click 'Reject all non-essential cookies' only necessary cookies providing core functionality such as security, network management, and accessibility will be enabled. Click 'Find out more' for information on how to change your cookie settings.

Epistasis is fast becoming central to the understanding of the complex relationship between genotype and phenotype observed in autoimmune disease. A study in this issue of the European Journal of Immunology uses in-depth analysis of genome-wide mapping by polymorphic microsatellite markers to shed light on the genomic control of autoimmunity and self-tolerance.

Original publication

DOI

10.1002/eji.201141811

Type

Journal article

Journal

European journal of immunology

Publication Date

08/2011

Volume

41

Pages

2152 - 2154

Addresses

Department of Genetics, UCL Institute of Ophthalmology, London, UK.

Keywords

Animals, Mice, Knockout, Cattle, Mice, Autoimmune Diseases, Genetic Predisposition to Disease, gamma-Globulins, Receptors, Cell Surface, Receptors, IgG, Antigens, CD, Immune Tolerance, Epistasis, Genetic, Signaling Lymphocytic Activation Molecule Family Member 1