Skip to main content

Cookies on this website

We use cookies to ensure that we give you the best experience on our website. If you click 'Accept all cookies' we'll assume that you are happy to receive all cookies and you won't see this message again. If you click 'Reject all non-essential cookies' only necessary cookies providing core functionality such as security, network management, and accessibility will be enabled. Click 'Find out more' for information on how to change your cookie settings.

Colonization of the intestinal lumen precedes invasive infection for a wide range of enteropathogenic and opportunistic pathogenic bacteria. We show that combining oral vaccination with engineered or selected niche-competitor strains permits pathogen exclusion and strain replacement in the mouse gut lumen. This approach can be applied either prophylactically to prevent invasion of nontyphoidal Salmonella strains, or therapeutically to displace an established Escherichia coli. Both intact adaptive immunity and metabolic niche competition are necessary for efficient vaccine-enhanced competition. Our findings imply that mucosal antibodies have evolved to work in the context of gut microbial ecology by influencing the outcome of competition. This has broad implications for the elimination of pathogenic and antibiotic-resistant bacterial reservoirs and for rational microbiota engineering.

Original publication

DOI

10.1126/science.adp5011

Type

Journal

Science

Publication Date

04/04/2025

Volume

388

Pages

74 - 81

Keywords

Animals, Mice, Escherichia coli, Salmonella Vaccines, Salmonella typhimurium, Immunity, Mucosal, Mice, Inbred C57BL, Administration, Oral, Escherichia coli Infections, Gastrointestinal Microbiome, Female, Adaptive Immunity, Antibiosis, Antibodies, Bacterial